MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability

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dc.contributor.authorEphraums, James
dc.contributor.authorYoukhana, Janet
dc.contributor.authorRaina, Aparna S.
dc.contributor.authorSchulstad, Grace
dc.contributor.authorCroft, Kento
dc.contributor.authorMawson, Amanda
dc.contributor.authorKokkinos, John
dc.contributor.authorGonzales\\-Aloy, Estrella
dc.contributor.authorIgnacio, Rosa Mistica C.
dc.contributor.authorMcCarroll, Joshua A.
dc.contributor.authorBoyer, Cyrille
dc.contributor.authorGoldstein, David
dc.contributor.authorPajic, Marina
dc.contributor.authorHaghighi, Koroush S.
dc.contributor.authorJohns, Amber
dc.contributor.authorGill, Anthony J.
dc.contributor.authorErkan, Mert
dc.contributor.authorPhillips, Phoebe A.
dc.contributor.authorSharbeen, George
dc.contributor.authorAustralian Pancreatic Canc Genome Initiative APGI
dc.date.accessioned2025-10-16T15:12:03Z
dc.date.issued2025
dc.identifier.doi10.1016/j.neo.2025.101138
dc.identifier.otherWOS:001426315500001
dc.identifier.urihttps://openaccess.acibadem.edu.tr/handle/11443/5211
dc.publisherELSEVIER SCIENCE INC
dc.sourceNEOPLASIA
dc.subjectPancreatic cancer
dc.subjectChemosensitisation
dc.subjectOxidative stress
dc.subjectDNA repair
dc.titleMYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability
dc.typeArticle

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